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Gap junction-mediated intercellular biochemical coupling in cochlear supporting cells is required for normal cochlear functions

Dysfunction of gap junctions (GJs) caused by mutations in connexin26 (Cx26) and Cx30 accounts for nearly half of all cases of hereditary nonsyndromic deafness cases. Although it is widely held that GJs connecting supporting cells in the organ of Corti mainly provide ionic pathways for rapid removal...

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Detalles Bibliográficos
Main Authors: Zhang, Yanping, Tang, Wenxue, Ahmad, Shoab, Sipp, James A., Chen, Ping, Lin, Xi
Formato: Artigo
Idioma:Inglês
Publicado: National Academy of Sciences 2005
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC1257692/
https://ncbi.nlm.nih.gov/pubmed/16217030
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0501859102
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