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Gap junction-mediated intercellular biochemical coupling in cochlear supporting cells is required for normal cochlear functions

Dysfunction of gap junctions (GJs) caused by mutations in connexin26 (Cx26) and Cx30 accounts for nearly half of all cases of hereditary nonsyndromic deafness cases. Although it is widely held that GJs connecting supporting cells in the organ of Corti mainly provide ionic pathways for rapid removal...

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Autors principals: Zhang, Yanping, Tang, Wenxue, Ahmad, Shoab, Sipp, James A., Chen, Ping, Lin, Xi
Format: Artigo
Idioma:Inglês
Publicat: National Academy of Sciences 2005
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC1257692/
https://ncbi.nlm.nih.gov/pubmed/16217030
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0501859102
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