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Gap junction-mediated intercellular biochemical coupling in cochlear supporting cells is required for normal cochlear functions
Dysfunction of gap junctions (GJs) caused by mutations in connexin26 (Cx26) and Cx30 accounts for nearly half of all cases of hereditary nonsyndromic deafness cases. Although it is widely held that GJs connecting supporting cells in the organ of Corti mainly provide ionic pathways for rapid removal...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2005
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1257692/ https://ncbi.nlm.nih.gov/pubmed/16217030 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0501859102 |
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