Mechanism of the regulation of type IB phosphoinositide 3OH-kinase byG-protein betagamma subunits.

Type IB phosphoinositide 3OH-kinase (PI3K) is activated by G-protein betagamma subunits (Gbetagammas). The enzyme is soluble and largely cytosolic in vivo. Its substrate, PtdIns(4,5)P(2), and the Gbetagammas are localized at the plasma membrane. We have addressed the mechanism by which Gbetagammas r...

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Autors principals: Krugmann, Sonja, Cooper, Matthew A, Williams, Dudley H, Hawkins, Phillip T, Stephens, Len R
Format: Artigo
Idioma:Inglês
Publicat: 2002
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Research Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC1222438/
https://ncbi.nlm.nih.gov/pubmed/11879201
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