Activation of c-Jun N-terminal kinase promotes survival of cardiac myocytes after oxidative stress.

Reperfusion injury occurs when ischaemic tissue is reperfused. It involves the generation and release of reactive oxygen that activates numerous signalling pathways and initiates cell death. Exposure of isolated cardiac myocytes to chronic hypoxia followed by reoxygenation results in the early activ...

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Hauptverfasser: Dougherty, Christopher J, Kubasiak, Lori A, Prentice, Howard, Andreka, Peter, Bishopric, Nanette H, Webster, Keith A
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2002
Schlagworte:
Research Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1222419/
https://ncbi.nlm.nih.gov/pubmed/11879182
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