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Cell cycle inhibition provides neuroprotection and reduces glial proliferation and scar formation after traumatic brain injury
Traumatic brain injury (TBI) causes neuronal apoptosis, inflammation, and reactive astrogliosis, which contribute to secondary tissue loss, impaired regeneration, and associated functional disabilities. Here, we show that up-regulation of cell cycle components is associated with caspase-mediated neu...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2005
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1149422/ https://ncbi.nlm.nih.gov/pubmed/15923260 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0500989102 |
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