Is the inherited ACE genotype a trump or a joker?

Por Mitch, W E
Publicado em 1995

Mechanisms accelerating muscle atrophy in catabolic diseases.

Por Mitch, W. E.
Publicado em 2000

Muscle protein turnover and glucose uptake in acutely uremic rats. Effects of insulin and the duration of renal insufficiency.

Por Clark, A S, Mitch, W E
Publicado em 1983

Specificity of the effects of leucine and its metabolites on protein degradation in skeletal muscle.

Por Mitch, W E, Clark, A S
Publicado em 1984

Comparison of protein synthesis and degradation in incubated and perfused muscle.

Por Clark, A S, Mitch, W E
Publicado em 1983

Nitrogen sparing induced by leucine compared with that induced by its keto analogue, alpha-ketoisocaproate, in fasting obese man.

Por Mitch, W E, Walser, M, Sapir, D G
Publicado em 1981

Systemic response to thermal injury in rats. Accelerated protein degradation and altered glucose utilization in muscle.

Por Clark, A S, Kelly, R A, Mitch, W E
Publicado em 1984

Selectivity of the insulin-like actions of vanadate on glucose and protein metabolism in skeletal muscle.

Por Clark, A S, Fagan, J M, Mitch, W E
Publicado em 1985

Metabolic acidosis stimulates protein degradation in rat muscle by a glucocorticoid-dependent mechanism.

Por May, R C, Kelly, R A, Mitch, W E
Publicado em 1986

Mechanisms for defects in muscle protein metabolism in rats with chronic uremia. Influence of metabolic acidosis.

Por May, R C, Kelly, R A, Mitch, W E
Publicado em 1987

Protein degradation and increased mRNAs encoding proteins of the ubiquitin-proteasome proteolytic pathway in BC3H1 myocytes require an interaction between glucocorticoids and acidi...

Por Isozaki, U, Mitch, W E, England, B K, Price, S R
Publicado em 1996

Abnormal cation transport in uremia. Mechanisms in adipocytes and skeletal muscle from uremic rats.

Por Druml, W, Kelly, R A, May, R C, Mitch, W E
Publicado em 1988

Reversible captopril-associated bone marrow aplasia.

Por Strair, R K, Mitch, W E, Faller, D V, Skorecki, K L
Publicado em 1985

The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway.

Por Bailey, J L, Wang, X, England, B K, Price, S R, Ding, X, Mitch, W E
Publicado em 1996

Dichloroacetate inhibits glycolysis and augments insulin-stimulated glycogen synthesis in rat muscle.

Por Clark, A S, Mitch, W E, Goodman, M N, Fagan, J M, Goheer, M A, Curnow, R T
Publicado em 1987

Interactions between L-arginine and L-glutamine change endothelial NO production. An effect independent of NO synthase substrate availability.

Por Arnal, J F, Münzel, T, Venema, R C, James, N L, Bai, C L, Mitch, W E, Harrison, D G
Publicado em 1995

Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription.

Por Price, S R, Bailey, J L, Wang, X, Jurkovitz, C, England, B K, Ding, X, Phillips, L S, Mitch, W E
Publicado em 1996

Metabolic acidosis stimulates muscle protein degradation by activating the adenosine triphosphate-dependent pathway involving ubiquitin and proteasomes.

Por Mitch, W E, Medina, R, Grieber, S, May, R C, England, B K, Price, S R, Bailey, J L, Goldberg, A L
Publicado em 1994

Glomerular function in Pima Indians with noninsulin-dependent diabetes mellitus of recent onset.

Por Myers, B D, Nelson, R G, Williams, G W, Bennett, P H, Hardy, S A, Berg, R L, Loon, N, Knowler, W C, Mitch, W E
Publicado em 1991