A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy

od Moreau, Adrien, Gosselin-Badaroudine, Pascal, Mercier, Aurélie, Burger, Bettina, Keller, Dagmar I., Chahine, Mohamed
izdano v Sci Rep (2018)

iPSC-derived cardiomyocytes from patients with myotonic dystrophy type 1 have abnormal ion channel functions and slower conduction velocities

od Poulin, Hugo, Mercier, Aurélie, Djemai, Mohammed, Pouliot, Valérie, Deschenes, Isabelle, Boutjdir, Mohamed, Puymirat, Jack, Chahine, Mohamed
izdano v Sci Rep (2021)

The β(1)-Subunit of Na(v)1.5 Cardiac Sodium Channel Is Required for a Dominant Negative Effect through α-α Interaction

od Mercier, Aurélie, Clément, Romain, Harnois, Thomas, Bourmeyster, Nicolas, Faivre, Jean-François, Findlay, Ian, Chahine, Mohamed, Bois, Patrick, Chatelier, Aurélien
Izdano 2012

Unmasked Brugada Pattern by Ajmaline Challenge in Patients with Myotonic Dystrophy Type 1

od Pambrun, Thomas, Bortone, Agustín, Bois, Patrick, Degand, Bruno, Patri, Sylvie, Mercier, Aurélie, Chahine, Mohamed, Chatelier, Aurélien, Coisne, Damien, Amiel, Alain
izdano v Ann Noninvasive Electrocardiol (2014)

A distinct de novo expression of Na(v)1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts

od Chatelier, Aurélien, Mercier, Aurélie, Tremblier, Boris, Thériault, Olivier, Moubarak, Majed, Benamer, Najate, Corbi, Pierre, Bois, Patrick, Chahine, Mohamed, Faivre, Jean François
Izdano 2012