A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy

Por Moreau, Adrien, Gosselin-Badaroudine, Pascal, Mercier, Aurélie, Burger, Bettina, Keller, Dagmar I., Chahine, Mohamed
Publicado no Sci Rep (2018)

iPSC-derived cardiomyocytes from patients with myotonic dystrophy type 1 have abnormal ion channel functions and slower conduction velocities

Por Poulin, Hugo, Mercier, Aurélie, Djemai, Mohammed, Pouliot, Valérie, Deschenes, Isabelle, Boutjdir, Mohamed, Puymirat, Jack, Chahine, Mohamed
Publicado no Sci Rep (2021)

The β(1)-Subunit of Na(v)1.5 Cardiac Sodium Channel Is Required for a Dominant Negative Effect through α-α Interaction

Por Mercier, Aurélie, Clément, Romain, Harnois, Thomas, Bourmeyster, Nicolas, Faivre, Jean-François, Findlay, Ian, Chahine, Mohamed, Bois, Patrick, Chatelier, Aurélien
Publicado em 2012

Unmasked Brugada Pattern by Ajmaline Challenge in Patients with Myotonic Dystrophy Type 1

Por Pambrun, Thomas, Bortone, Agustín, Bois, Patrick, Degand, Bruno, Patri, Sylvie, Mercier, Aurélie, Chahine, Mohamed, Chatelier, Aurélien, Coisne, Damien, Amiel, Alain
Publicado no Ann Noninvasive Electrocardiol (2014)

A distinct de novo expression of Na(v)1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts

Por Chatelier, Aurélien, Mercier, Aurélie, Tremblier, Boris, Thériault, Olivier, Moubarak, Majed, Benamer, Najate, Corbi, Pierre, Bois, Patrick, Chahine, Mohamed, Faivre, Jean François
Publicado em 2012