A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy

di Moreau, Adrien, Gosselin-Badaroudine, Pascal, Mercier, Aurélie, Burger, Bettina, Keller, Dagmar I., Chahine, Mohamed
Pubblicato in Sci Rep (2018)

iPSC-derived cardiomyocytes from patients with myotonic dystrophy type 1 have abnormal ion channel functions and slower conduction velocities

di Poulin, Hugo, Mercier, Aurélie, Djemai, Mohammed, Pouliot, Valérie, Deschenes, Isabelle, Boutjdir, Mohamed, Puymirat, Jack, Chahine, Mohamed
Pubblicato in Sci Rep (2021)

The β(1)-Subunit of Na(v)1.5 Cardiac Sodium Channel Is Required for a Dominant Negative Effect through α-α Interaction

di Mercier, Aurélie, Clément, Romain, Harnois, Thomas, Bourmeyster, Nicolas, Faivre, Jean-François, Findlay, Ian, Chahine, Mohamed, Bois, Patrick, Chatelier, Aurélien
Pubblicazione 2012

Unmasked Brugada Pattern by Ajmaline Challenge in Patients with Myotonic Dystrophy Type 1

di Pambrun, Thomas, Bortone, Agustín, Bois, Patrick, Degand, Bruno, Patri, Sylvie, Mercier, Aurélie, Chahine, Mohamed, Chatelier, Aurélien, Coisne, Damien, Amiel, Alain
Pubblicato in Ann Noninvasive Electrocardiol (2014)

A distinct de novo expression of Na(v)1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts

di Chatelier, Aurélien, Mercier, Aurélie, Tremblier, Boris, Thériault, Olivier, Moubarak, Majed, Benamer, Najate, Corbi, Pierre, Bois, Patrick, Chahine, Mohamed, Faivre, Jean François
Pubblicazione 2012