A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy

gan Moreau, Adrien, Gosselin-Badaroudine, Pascal, Mercier, Aurélie, Burger, Bettina, Keller, Dagmar I., Chahine, Mohamed
Cyhoeddwyd yn Sci Rep (2018)

iPSC-derived cardiomyocytes from patients with myotonic dystrophy type 1 have abnormal ion channel functions and slower conduction velocities

gan Poulin, Hugo, Mercier, Aurélie, Djemai, Mohammed, Pouliot, Valérie, Deschenes, Isabelle, Boutjdir, Mohamed, Puymirat, Jack, Chahine, Mohamed
Cyhoeddwyd yn Sci Rep (2021)

The β(1)-Subunit of Na(v)1.5 Cardiac Sodium Channel Is Required for a Dominant Negative Effect through α-α Interaction

gan Mercier, Aurélie, Clément, Romain, Harnois, Thomas, Bourmeyster, Nicolas, Faivre, Jean-François, Findlay, Ian, Chahine, Mohamed, Bois, Patrick, Chatelier, Aurélien
Cyhoeddwyd 2012

Unmasked Brugada Pattern by Ajmaline Challenge in Patients with Myotonic Dystrophy Type 1

gan Pambrun, Thomas, Bortone, Agustín, Bois, Patrick, Degand, Bruno, Patri, Sylvie, Mercier, Aurélie, Chahine, Mohamed, Chatelier, Aurélien, Coisne, Damien, Amiel, Alain
Cyhoeddwyd yn Ann Noninvasive Electrocardiol (2014)

A distinct de novo expression of Na(v)1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts

gan Chatelier, Aurélien, Mercier, Aurélie, Tremblier, Boris, Thériault, Olivier, Moubarak, Majed, Benamer, Najate, Corbi, Pierre, Bois, Patrick, Chahine, Mohamed, Faivre, Jean François
Cyhoeddwyd 2012