Plasma and urinary p21: potential biomarkers of AKI and renal aging

Por Johnson, Ali C., Zager, Richard A.
Publicado no Am J Physiol Renal Physiol (2018)

Renal ischemia-reperfusion injury upregulates histone-modifying enzyme systems and alters histone expression at proinflammatory/profibrotic genes

Por Zager, Richard A., Johnson, Ali C. M.
Publicado em 2009

Progressive histone alterations and proinflammatory gene activation: consequences of heme protein/iron-mediated proximal tubule injury

Por Zager, Richard A., Johnson, Ali C. M.
Publicado em 2010

Mechanisms Underlying Increased TIMP2 and IGFBP7 Urinary Excretion in Experimental AKI

Por Johnson, Ali C.M., Zager, Richard A.
Publicado no J Am Soc Nephrol (2018)

Acute kidney injury induces dramatic p21 upregulation via a novel, glucocorticoid-activated, pathway

Por Zager, Richard A., Johnson, Ali C. M.
Publicado no Am J Physiol Renal Physiol (2019)

The NRF2 stimulating agent, tin protoporphyrin, activates protective cytokine pathways in healthy human subjects and in patients with chronic kidney disease

Por Zager, Richard A., Johnson, Ali C. M.
Publicado no Physiol Rep (2020)

Post-ischemic azotemia as a partial ‘brake’, slowing progressive kidney disease

Por Zager, Richard A., Johnson, Ali C., Becker, Kirsten
Publicado em 2013

Uremia impacts renal inflammatory cytokine gene expression in the setting of experimental acute kidney injury

Por Zager, Richard A., Johnson, Ali C. M., Lund, Steve
Publicado em 2009

Acute unilateral ischemic renal injury induces progressive renal inflammation, lipid accumulation, histone modification, and “end-stage” kidney disease

Por Zager, Richard A., Johnson, Ali C. M., Becker, Kirsten
Publicado em 2011

Plasma and Urinary Heme Oxygenase-1 in AKI

Por Zager, Richard A., Johnson, Ali C.M., Becker, Kirsten
Publicado em 2012

Proximal tubule haptoglobin gene activation is an integral component of the acute kidney injury “stress response”

Por Zager, Richard A., Vijayan, Anitha, Johnson, Ali C. M.
Publicado em 2012

Renal cortical hemopexin accumulation in response to acute kidney injury

Por Zager, Richard A., Johnson, Ali C. M., Becker, Kirsten
Publicado em 2012

Renal Cortical Lactate Dehydrogenase: A Useful, Accurate, Quantitative Marker of In Vivo Tubular Injury and Acute Renal Failure

Por Zager, Richard A., Johnson, Ali C. M., Becker, Kirsten
Publicado em 2013

Renal Cortical Pyruvate Depletion during AKI

Por Zager, Richard A., Johnson, Ali C.M., Becker, Kirsten
Publicado em 2014

Renal cortical albumin gene induction and urinary albumin excretion in response to acute kidney injury

Por Ware, Lorraine B., Johnson, Ali C. M., Zager, Richard A.
Publicado em 2011

Rapid Renal Alpha-1 Antitrypsin Gene Induction in Experimental and Clinical Acute Kidney Injury

Por Zager, Richard A., Johnson, Ali C. M., Frostad, Kirsten B.
Publicado em 2014

Acute hepatic ischemic-reperfusion injury induces a renal cortical “stress response,” renal “cytoresistance,” and an endotoxin hyperresponsive state

Por Zager, Richard A., Johnson, Ali C. M., Frostad, Kirsten B.
Publicado em 2014

Acute Tubular Injury Causes Dysregulation of Cellular Cholesterol Transport Proteins

Por Zager, Richard A., Johnson, Ali C.M., Hanson, Sherry Y., Shah, Vallabh O.
Publicado em 2003

A Pharmacologic “Stress Test” for Assessing Select Antioxidant Defenses in Patients with CKD

Por Zager, Richard A., Johnson, Ali C.M., Guillem, Alvaro, Keyser, Jeff, Singh, Bhupinder
Publicado no Clin J Am Soc Nephrol (2020)

Experimental Glomerulopathy Alters Renal Cortical Cholesterol, SR-B1, ABCA1, and HMG CoA Reductase Expression

Por Johnson, Ali C. M., Yabu, Julie M., Hanson, Sherry, Shah, Vallabh O., Zager, Richard A.
Publicado em 2003