טוען...
Post-ischemic azotemia as a partial ‘brake’, slowing progressive kidney disease
BACKGROUND: Recent experimental work suggests a paradox: although uremia evokes systemic toxicities, in the setting of AKI, it can induce intrarenal cytoprotective and anti-inflammatory effects. Whether these influences can attenuate post-ischemic kidney disease progression remains unknown. METHODS:...
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| Main Authors: | , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Oxford University Press
2013
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3685309/ https://ncbi.nlm.nih.gov/pubmed/23543590 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/ndt/gft040 |
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