Targeting synaptic pathology in multiple sclerosis: fingolimod to the rescue?

Por Gillingwater, Thomas H
Publicado em 2012

Compartmental neurodegeneration and synaptic plasticity in the Wld(s) mutant mouse

Por Gillingwater, Thomas H, Ribchester, Richard R
Publicado em 2001

Selective loss of alpha motor neurons with sparing of gamma motor neurons and spinal cord cholinergic neurons in a mouse model of spinal muscular atrophy

Por Powis, Rachael A., Gillingwater, Thomas H.
Publicado no J Anat (2015)

Dissection of the Transversus Abdominis Muscle for Whole-mount Neuromuscular Junction Analysis

Por Murray, Lyndsay, Gillingwater, Thomas H, Kothary, Rashmi
Publicado em 2014

UBA1: At the Crossroads of Ubiquitin Homeostasis and Neurodegeneration

Por Groen, Ewout J.N., Gillingwater, Thomas H.
Publicado no Trends Mol Med (2015)

Rapid loss of motor nerve terminals following hypoxia–reperfusion injury occurs via mechanisms distinct from classic Wallerian degeneration

Por Baxter, Becki, Gillingwater, Thomas H, Parson, Simon H
Publicado em 2008

The contribution of mouse models to understanding the pathogenesis of spinal muscular atrophy

Por Sleigh, James N., Gillingwater, Thomas H., Talbot, Kevin
Publicado em 2011

Eldred Wright Walls, MD, FRCS, FRCSE, FRSE

Por Findlater, Gordon S, Gillingwater, Thomas H, Kristmundsdottir, Fanney, Parson, Simon H
Publicado em 2008

Overview of Current Drugs and Molecules in Development for Spinal Muscular Atrophy Therapy

Por Shorrock, Hannah K., Gillingwater, Thomas H., Groen, Ewout J. N.
Publicado no Drugs (2018)

Molecular Mechanisms Underlying Sensory-Motor Circuit Dysfunction in SMA

Por Shorrock, Hannah K., Gillingwater, Thomas H., Groen, Ewout J. N.
Publicado no Front Mol Neurosci (2019)

Ultrastructural correlates of synapse withdrawal at axotomized neuromuscular junctions in mutant and transgenic mice expressing the Wld gene

Por Gillingwater, Thomas H, Ingham, Cali A, Coleman, Michael P, Ribchester, Richard R
Publicado em 2003

Synaptic Protection in the Brain of Wld(S) Mice Occurs Independently of Age but Is Sensitive to Gene-Dose

Por Wright, Ann K., Wishart, Thomas M., Ingham, Cali A., Gillingwater, Thomas H.
Publicado em 2010

Morphological analysis of neuromuscular junction development and degeneration in rodent lumbrical muscles

Por Sleigh, James N., Burgess, Robert W., Gillingwater, Thomas H., Cader, M. Zameel
Publicado em 2014

The role of survival motor neuron protein (SMN) in protein homeostasis

Por Chaytow, Helena, Huang, Yu-Ting, Gillingwater, Thomas H., Faller, Kiterie M. E.
Publicado no Cell Mol Life Sci (2018)

Loss of translation elongation factor (eEF1A2) expression in vivo differentiates between Wallerian degeneration and dying-back neuronal pathology

Por Murray, Lyndsay M, Thomson, Derek, Conklin, Annalijn, Wishart, Thomas M, Gillingwater, Thomas H
Publicado em 2008

Using induced pluripotent stem cells (iPSC) to model human neuromuscular connectivity: promise or reality?

Por Thomson, Sophie R, Wishart, Thomas M, Patani, Rickie, Chandran, Siddharthan, Gillingwater, Thomas H
Publicado em 2012

mGluR5 Regulates Glutamate-Dependent Development of the Mouse Somatosensory Cortex

Por Wijetunge, Lasani S., Till, Sally M., Gillingwater, Thomas H., Ingham, Cali A., Kind, Peter C.
Publicado em 2008

Label-free proteomics identifies Calreticulin and GRP75/Mortalin as peripherally accessible protein biomarkers for spinal muscular atrophy

Por Mutsaers, Chantal A, Lamont, Douglas J, Hunter, Gillian, Wishart, Thomas M, Gillingwater, Thomas H
Publicado em 2013

Developmental and degenerative cardiac defects in the Taiwanese mouse model of severe spinal muscular atrophy

Por Maxwell, Gillian K., Szunyogova, Eva, Shorrock, Hannah K., Gillingwater, Thomas H., Parson, Simon H.
Publicado no J Anat (2018)

Protein Product of CLN6 Gene Responsible for Variant Late-Onset Infantile Neuronal Ceroid Lipofuscinosis Interacts with CRMP-2

Por Benedict, Jared W., Getty, Amanda L., Wishart, Thomas M., Gillingwater, Thomas H., Pearce, David A.
Publicado em 2009