A Kir6.2 Pore Mutation Causes Inactivation of ATP-Sensitive Potassium Channels by Disrupting PIP(2)-Dependent Gating

Por Bushman, Jeremy D., Zhou, Qing, Shyng, Show-Ling
Publicado em 2013

A proton current associated with sour taste: distribution and functional properties

Por Bushman, Jeremy D., Ye, Wenlei, Liman, Emily R.
Publicado no FASEB J (2015)

Characterization and Functional Restoration of a Potassium Channel Kir6.2 Pore Mutation Identified in Congenital Hyperinsulinism

Por Bushman, Jeremy D., Gay, Joel W., Tewson, Paul, Stanley, Charles A., Shyng, Show-Ling
Publicado em 2010

Optimized expression vector for ion channel studies in Xenopus oocytes and mammalian cells using alfalfa mosaic virus

Por Venkatachalan, Srinivasan P., Bushman, Jeremy D., Mercado, José L., Sancar, Feyza, Christopherson, Kelly R., Boileau, Andrew J.
Publicado em 2006

Destabilization of ATP-sensitive Potassium Channel Activity by Novel KCNJ11 Mutations Identified in Congenital Hyperinsulinism

Por Lin, Yu-Wen, Bushman, Jeremy D., Yan, Fei-Fei, Haidar, Sara, MacMullen, Courtney, Ganguly, Arupa, Stanley, Charles A., Shyng, Show-Ling
Publicado em 2008

The K(+) channel K(IR)2.1 functions in tandem with proton influx to mediate sour taste transduction

Por Ye, Wenlei, Chang, Rui B., Bushman, Jeremy D., Tu, Yu-Hsiang, Mulhall, Eric M., Wilson, Courtney E., Cooper, Alexander J., Chick, Wallace S., Hill-Eubanks, David C., Nelson, Mark T., Kinnamon, Sue C., Liman, Emily R.
Publicado no Proc Natl Acad Sci U S A (2016)