Clostridium botulinum C2 Toxin Delays Entry into Mitosis and Activation of p34(cdc2) Kinase and cdc25-C Phosphatase in HeLa cells
The Clostridium botulinum C2 toxin ADP-ribosylates monomeric actin, thereby inducing disassembly of actin filaments, alteration of focal adhesions, and rounding of cells. After treatment with C2 toxin, cells stop to proliferate but remain viable for about 2 days. In view of reported correlations bet...
שמור ב:
| הוצא לאור ב: | Infect Immun |
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| Principais autores: | , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Microbiology (ASM)
1999
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC96856/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10496881/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.67.10.5083-5090.1999 |
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