A Calcineurin-NFATc3-Dependent Pathway Regulates Skeletal Muscle Differentiation and Slow Myosin Heavy-Chain Expression

Lignende værker

• Targeted Disruption of NFATc3, but Not NFATc4, Reveals an Intrinsic Defect in Calcineurin-Mediated Cardiac Hypertrophic Growth
  af: Wilkins, Benjamin J., et al.
  Udgivet: (2002)
• Akirin2 promotes slow myosin heavy chain expression by CaN/NFATc1 signaling in porcine skeletal muscle satellite cells
  af: Chen, Xiaoling, et al.
  Udgivet: (2017)
• Calcineurin plays a modulatory role in loading-induced regulation of type I myosin heavy chain gene expression in slow skeletal muscle
  af: Pandorf, Clay E., et al.
  Udgivet: (2009)
• Novel epigenetic regulation of skeletal muscle myosin heavy chain genes. Focus on “Differential epigenetic modifications of histones at the myosin heavy chain genes in fast and slow skeletal muscle fibers and in response to muscle unloading”
  af: Zwetsloot, Kevin A., et al.
  Udgivet: (2009)
• Regulation of expression of avian slow myosin heavy-chain isoforms.
  af: Reid, S K, et al.
  Udgivet: (1989)