Increased fibroblast accumulation in the equine heart following persistent atrial fibrillation

BACKGROUND: Fibroblasts maintain the extracellular matrix homeostasis and may couple to cardiomyocytes through gap junctions and thereby increase the susceptibility to slow conduction and cardiac arrhythmias, such as atrial fibrillation (AF). In this study, we used an equine model of persistent AF t...

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Dettagli Bibliografici
Pubblicato in:Int J Cardiol Heart Vasc
Autori principali: Saljic, Arnela, Friederike Fenner, Merle, Winters, Joris, Flethøj, Mette, Eggert Eggertsen, Caroline, Carstensen, Helena, Dalgas Nissen, Sarah, Melis Hesselkilde, Eva, van Hunnik, Arne, Schotten, Ulrich, Sørensen, Ulrik, Jespersen, Thomas, Verheule, Sander, Buhl, Rikke
Natura: Artigo
Lingua:Inglês
Pubblicazione: Elsevier 2021
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Original Paper
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC8322305/
https://ncbi.nlm.nih.gov/pubmed/34355058
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ijcha.2021.100842
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