Lack of APOL1 in proximal tubules of normal human kidneys and proteinuric APOL1 transgenic mouse kidneys

The mechanism of pathogenesis associated with APOL1 polymorphisms and risk for non-diabetic chronic kidney disease (CKD) is not fully understood. Prior studies have minimized a causal role for the circulating APOL1 protein, thus efforts to understand kidney pathogenesis have focused on APOL1 express...

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Detaylı Bibliyografya
Yayımlandı:PLoS One
Asıl Yazarlar: Blessing, Natalya A., Wu, Zhenzhen, Madhavan, Sethu M., Choy, Jonathan W., Chen, Michelle, Shin, Myung K., Hoek, Maarten, Sedor, John R., O’Toole, John F., Bruggeman, Leslie A.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Public Library of Science 2021
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC8211208/
https://ncbi.nlm.nih.gov/pubmed/34138902
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0253197
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