Increased cerebral mitochondrial dysfunction and reactive oxygen species with cardiopulmonary bypass

 : OBJECTIVES: Neurodevelopmental injury after cardiac surgery using cardiopulmonary bypass (CPB) for congenital heart defects is common, but the mechanism behind this injury is unclear. This study examines the impact of CPB on cerebral mitochondrial reactive oxygen species (ROS) generation and mito...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Eur J Cardiothorac Surg
Päätekijät: Volk, Lindsay E, Mavroudis, Constantine D, Ko, Tiffany, Hallowell, Thomas, Delso, Nile, Roberts, Anna L, Starr, Jonathan, Landis, William, Lin, Yuxi, Hefti, Marco, Morgan, Ryan W, Melchior, Richard W, Rosenthal, Tami M, Chappell, Alexander, Fisher, Douglas, Dreher, Molly, Licht, Daniel J, Chen, Jonathan, Gaynor, J. William, Mascio, Christopher E, Kilbaugh, Todd J
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Oxford University Press 2020
Aiheet:
Experimental
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC8203249/
https://ncbi.nlm.nih.gov/pubmed/33367535
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/ejcts/ezaa439
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