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Increased cerebral mitochondrial dysfunction and reactive oxygen species with cardiopulmonary bypass
: OBJECTIVES: Neurodevelopmental injury after cardiac surgery using cardiopulmonary bypass (CPB) for congenital heart defects is common, but the mechanism behind this injury is unclear. This study examines the impact of CPB on cerebral mitochondrial reactive oxygen species (ROS) generation and mito...
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| 出版年: | Eur J Cardiothorac Surg |
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| 主要な著者: | , , , , , , , , , , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Oxford University Press
2020
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8203249/ https://ncbi.nlm.nih.gov/pubmed/33367535 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/ejcts/ezaa439 |
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