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Epigallocatechin-3-gallate modulates Tau Post-translational modifications and cytoskeletal network
Background: Alzheimer’s disease is a type of dementia denoted by progressive neuronal death due to the accumulation of proteinaceous aggregates of Tau. Post-translational modifications like hyperphosphorylation, truncation, glycation, etc. play a pivotal role in Tau pathogenesis. Glycation of Tau ai...
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| Vydáno v: | Oncotarget |
|---|---|
| Hlavní autoři: | , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Impact Journals LLC
2021
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8169072/ https://ncbi.nlm.nih.gov/pubmed/34084282 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.27963 |
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