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TRPC channel-derived calcium fluxes differentially regulate ATP and flow–induced activation of eNOS
Endothelial dysfunction, characterised by impaired nitric oxide (NO) bioavailability, arises in response to a variety of cardiovascular risk factors and precedes atherosclerosis. NO is produced by tight regulation of endothelial nitric oxide synthase (eNOS) activity in response to vasodilatory stimu...
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| Published in: | Nitric Oxide |
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| Main Authors: | , , , |
| Format: | Artigo |
| Language: | Inglês |
| Published: |
2021
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| Subjects: | |
| Online Access: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8145791/ https://ncbi.nlm.nih.gov/pubmed/33813098 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.niox.2021.03.005 |
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