KCNK3 Mutation Causes Altered Immune Function in Pulmonary Arterial Hypertension Patients and Mouse Models

Loss of function KCNK3 mutation is one of the gene variants driving hereditary pulmonary arterial hypertension (PAH). KCNK3 is expressed in several cell and tissue types on both membrane and endoplasmic reticulum and potentially plays a role in multiple pathological process associated with PAH. Howe...

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Detaylı Bibliyografya
Yayımlandı:Int J Mol Sci
Asıl Yazarlar: West, James D., Austin, Eric D., Rizzi, Elise M., Yan, Ling, Tanjore, Harikrishna, Crabtree, Amber L., Moore, Christy S., Muthian, Gladson, Carrier, Erica J., Jacobson, David A., Hamid, Rizwan, Kendall, Peggy L., Majka, Susan, Rathinasabapathy, Anandharajan
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: MDPI 2021
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Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC8126011/
https://ncbi.nlm.nih.gov/pubmed/34065088
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms22095014
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