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Atypical E2Fs either Counteract or Cooperate with RB during Tumorigenesis Depending on Tissue Context
SIMPLE SUMMARY: In virtually all human malignancies, the CDK-RB-E2F pathway is dysregulated resulting in the activation of the E2F transcriptional network. Rb and atypical E2Fs are the most important negative regulators of E2F-dependent transcription during tumorigenesis. However, it is unknown whet...
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| Publicado no: | Cancers (Basel) |
|---|---|
| Main Authors: | , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
MDPI
2021
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8122802/ https://ncbi.nlm.nih.gov/pubmed/33922435 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/cancers13092033 |
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