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Activation of GSK3 Prevents Termination of TNF-Induced Signaling
BACKGROUND: Termination of TNF-induced signaling plays a key role in the resolution of inflammation with dysregulations leading to severe pathophysiological conditions (sepsis, chronic inflammatory disease, cancer). Since a recent phospho-proteome analysis in human monocytes suggested GSK3 as a rele...
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| Gepubliceerd in: | J Inflamm Res |
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| Hoofdauteurs: | , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Dove
2021
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8111165/ https://ncbi.nlm.nih.gov/pubmed/33986607 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2147/JIR.S300806 |
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