Lactobacillus acidophilus Induces a Strain-specific and Toll-Like Receptor 2–Dependent Enhancement of Intestinal Epithelial Tight Junction Barrier and Protection Against Intestinal Inflammation

Lignende værker

• Matrix metalloproteinase 9-induced increase in intestinal epithelial tight junction permeability contributes to the severity of experimental DSS colitis
  af: Nighot, Prashant, et al.
  Udgivet: (2015)
• Lipopolysaccharide-Induced Increase in Intestinal Epithelial Tight Permeability Is Mediated by Toll-Like Receptor 4/Myeloid Differentiation Primary Response 88 (MyD88) Activation of Myosin Light Chain Kinase Expression
  af: Nighot, Meghali, et al.
  Udgivet: (2017)
• Lipopolysaccharide regulation of intestinal tight junction permeability is mediated by TLR-4 signal transduction pathway activation of FAK and MyD88
  af: Guo, Shuhong, et al.
  Udgivet: (2015)
• Occludin regulates macromolecule flux across the intestinal epithelial tight junction barrier
  af: Al-Sadi, Rana, et al.
  Udgivet: (2011)
• Mechanism of cytokine modulation of epithelial tight junction barrier
  af: Al-Sadi, Rana, et al.
  Udgivet: (2009)