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Lin28a ameliorates glucotoxicity-induced β-cell dysfunction and apoptosis
An excessive and prolonged increase in glucose levels causes β-cell dysregulation, which is accompanied by impaired insulin synthesis and secretion, a condition known as glucotoxicity. Although it is known that both Lin28a and Lin28b regulate glucose metabolism, other molecular mechanisms that may p...
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| Publicado no: | BMB Rep |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Korean Society for Biochemistry and Molecular Biology
2021
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8093937/ https://ncbi.nlm.nih.gov/pubmed/33691905 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.5483/BMBRep.2021.54.4.255 |
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