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Replicated chromatin curtails 53BP1 recruitment in BRCA1-proficient and BRCA1-deficient cells
DNA double-strand breaks can be repaired by non-homologous end-joining or homologous recombination. Which pathway is used depends on the balance between the tumor suppressors 53BP1 and BRCA1 and on the availability of an undamaged template DNA for homology-directed repair. How cells switch from a 53...
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| Vydáno v: | Life Sci Alliance |
|---|---|
| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Life Science Alliance LLC
2021
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8046418/ https://ncbi.nlm.nih.gov/pubmed/33811064 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.26508/lsa.202101023 |
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