Interleukin-19 Abrogates Experimental Autoimmune Encephalomyelitis by Attenuating Antigen-Presenting Cell Activation

Interleukin-19 (IL-19) acts as a negative-feedback regulator to limit proinflammatory response of macrophages and microglia in autocrine/paracrine manners in various inflammatory diseases. Multiple sclerosis (MS) is a major neuroinflammatory disease in the central nervous system (CNS), but it remain...

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Dettagli Bibliografici
Pubblicato in:Front Immunol
Autori principali: Horiuchi, Hiroshi, Parajuli, Bijay, Komiya, Hiroyasu, Ogawa, Yuki, Jin, Shijie, Takahashi, Keita, Azuma, Yasu-Taka, Tanaka, Fumiaki, Suzumura, Akio, Takeuchi, Hideyuki
Natura: Artigo
Lingua:Inglês
Pubblicazione: Frontiers Media S.A. 2021
Soggetti:
Immunology
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC7990911/
https://ncbi.nlm.nih.gov/pubmed/33776998
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fimmu.2021.615898
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