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TET-dioxygenase deficiency in oncogenesis and its targeting for tumor-selective therapeutics
TET2 is one of the most frequently mutated genes in myeloid neoplasms. TET2 loss-of-function perturbs myeloid differentiation and causes clonal expansion. Despite extensive knowledge regarding biochemical mechanisms underlying distorted myeloid differentiation, targeted therapies are lagging. Here w...
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| Publicat a: | Semin Hematol |
|---|---|
| Autors principals: | , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7938524/ https://ncbi.nlm.nih.gov/pubmed/33509440 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1053/j.seminhematol.2020.12.002 |
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