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Phosphorylation-dependent regulation of SPOP by LIMK2 promotes castration-resistant prostate cancer

BACKGROUND: SPOP, an E3 ubiquitin ligase adaptor, can act either as a tumour suppressor or a tumour promoter. In prostate cancer (PCa), it inhibits tumorigenesis by degrading several oncogenic substrates. SPOP is the most altered gene in PCa (~15%), which renders it ineffective, promoting cancer. Th...

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Detalhes bibliográficos
Publicado no:Br J Cancer
Main Authors: Nikhil, Kumar, Haymour, Hanan S., Kamra, Mohini, Shah, Kavita
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2020
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7921662/
https://ncbi.nlm.nih.gov/pubmed/33311589
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41416-020-01197-6
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