Mammalian Target of Rapamycin Signaling Enhances Ovalbumin-Induced Neutrophilic Airway Inflammation by Promoting Th17 Cell Polarization in Murine Noneosinophilic Asthma Model

Background: T helper 17 (Th17) is regarded as key immune cell in the pathogenesis of noneosinophilic asthma (NEA) due to the recruitment of neutrophils into the airways. The mammalian target of rapamycin (mTOR) is an important signaling molecule that plays a critical role in immune regulation. This...

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Gepubliceerd in:Pediatr Allergy Immunol Pulmonol
Hoofdauteurs: Wu, Jinhong, Zhong, Wenwei, Zhang, Hao, Yin, Yong
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: Mary Ann Liebert, Inc., publishers 2020
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Original Research
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7875112/
https://ncbi.nlm.nih.gov/pubmed/33406024
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/ped.2019.1088
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