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Exercise Training Stabilizes RyR2-Dependent Ca(2+) Release in Post-infarction Heart Failure

Aim: Dysfunction of the cardiac ryanodine receptor (RyR2) is an almost ubiquitous finding in animal models of heart failure (HF) and results in abnormal Ca(2+) release in cardiomyocytes that contributes to contractile impairment and arrhythmias. We tested whether exercise training (ET), as recommend...

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Bibliografske podrobnosti
izdano v:Front Cardiovasc Med
Main Authors: Danielsen, Tore Kristian, Sadredini, Mani, Manotheepan, Ravinea, Aronsen, Jan Magnus, Frisk, Michael, Hansen, Marie Haugsten, Andressen, Kjetil Wessel, Hougen, Karina, Levy, Finn Olav, Louch, William E., Sejersted, Ole Mathias, Sjaastad, Ivar, Stokke, Mathis Korseberg
Format: Artigo
Jezik:Inglês
Izdano: Frontiers Media S.A. 2021
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC7868397/
https://ncbi.nlm.nih.gov/pubmed/33569394
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fcvm.2020.623922
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