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Exercise Training Stabilizes RyR2-Dependent Ca(2+) Release in Post-infarction Heart Failure
Aim: Dysfunction of the cardiac ryanodine receptor (RyR2) is an almost ubiquitous finding in animal models of heart failure (HF) and results in abnormal Ca(2+) release in cardiomyocytes that contributes to contractile impairment and arrhythmias. We tested whether exercise training (ET), as recommend...
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| izdano v: | Front Cardiovasc Med |
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| Main Authors: | , , , , , , , , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
Frontiers Media S.A.
2021
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7868397/ https://ncbi.nlm.nih.gov/pubmed/33569394 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fcvm.2020.623922 |
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