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Loss of Slfn3 induces a sex-dependent repair vulnerability after 50% bowel resection
Bowel resection accelerates enterocyte proliferation in the remaining gut with suboptimal absorptive and digestive capacity because of a proliferation-associated decrease in functional differentiation markers. We hypothesized that although schlafen 3 (Slfn3) is an important regulator of enterocytic...
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| Publicat a: | Am J Physiol Gastrointest Liver Physiol |
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| Autors principals: | , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2021
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7864235/ https://ncbi.nlm.nih.gov/pubmed/33237796 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpgi.00344.2020 |
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