Loss of Slfn3 induces a sex-dependent repair vulnerability after 50% bowel resection

Bowel resection accelerates enterocyte proliferation in the remaining gut with suboptimal absorptive and digestive capacity because of a proliferation-associated decrease in functional differentiation markers. We hypothesized that although schlafen 3 (Slfn3) is an important regulator of enterocytic...

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Publicado en:Am J Physiol Gastrointest Liver Physiol
Autores principales: Vomhof-DeKrey, Emilie E., Lansing, Jack T., Darland, Diane C., Umthun, Josey, Stover, Allie D., Brown, Christopher, Basson, Marc D.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Physiological Society 2021
Materias:
Research Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC7864235/
https://ncbi.nlm.nih.gov/pubmed/33237796
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpgi.00344.2020
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