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The Newborn Fmr1 Knockout Mouse: A Novel Model of Excess Ubiquinone and Closed Mitochondrial Permeability Transition Pore in the Developing Heart
BACKGROUND: Mitochondrial permeability transition pore (mPTP) closure triggers cardiomyocyte differentiation during development while pathological opening causes cell death during myocardial ischemia-reperfusion and heart failure. Ubiquinone modulates the mPTP, however, little is known about its mec...
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| 出版年: | Pediatr Res |
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| 主要な著者: | , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2020
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7855053/ https://ncbi.nlm.nih.gov/pubmed/32674111 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41390-020-1064-6 |
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