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Desialylation of Atg5 by sialidase (Neu2) enhances autophagosome formation to induce anchorage-dependent cell death in ovarian cancer cells

Increased sialylation is one of the hallmarks of ovarian cancer (OC) but its relation with programmed cell death is not known. Here we explored the molecular interplay between autophagy, apoptosis/anoikis, and aberrant-expression of the PI3K-Akt/mTOR pathway in the context of sialidase. OC is accomp...

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Опубликовано в: :Cell Death Discov
Главные авторы: Satyavarapu, Eswara Murali, Nath, Shalini, Mandal, Chitra
Формат: Artigo
Язык:Inglês
Опубликовано: Nature Publishing Group UK 2021
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Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC7851153/
https://ncbi.nlm.nih.gov/pubmed/33526785
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41420-020-00391-y
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