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SAM68 promotes tumorigenesis in lung adenocarcinoma by regulating metabolic conversion via PKM alternative splicing

Background: A metabolic “switch” from oxidative phosphorylation to glycolysis provides tumor cells with energy and biosynthetic substrates, thereby promoting tumorigenesis and malignant progression. However, the mechanisms controlling this metabolic switch in tumors is not entirely clear. Methods: C...

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Dades bibliogràfiques
Publicat a:Theranostics
Autors principals: Zhu, Song, Chen, Weiping, Wang, Jizhong, Qi, Ling, Pan, Huilin, Feng, Zhengfu, Tian, Dongbo
Format: Artigo
Idioma:Inglês
Publicat: Ivyspring International Publisher 2021
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7847678/
https://ncbi.nlm.nih.gov/pubmed/33537092
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7150/thno.51360
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