The Bloom syndrome complex senses RPA-coated single-stranded DNA to restart stalled replication forks

The Bloom syndrome helicase BLM interacts with topoisomerase IIIα (TOP3A), RMI1 and RMI2 to form the BTR complex, which dissolves double Holliday junctions to produce non-crossover homologous recombination (HR) products. BLM also promotes DNA-end resection, restart of stalled replication forks, and...

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Publicat a:Nat Commun
Autors principals: Shorrocks, Ann-Marie K., Jones, Samuel E., Tsukada, Kaima, Morrow, Carl A., Belblidia, Zoulikha, Shen, Johanna, Vendrell, Iolanda, Fischer, Roman, Kessler, Benedikt M., Blackford, Andrew N.
Format: Artigo
Idioma:Inglês
Publicat: Nature Publishing Group UK 2021
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7838300/
https://ncbi.nlm.nih.gov/pubmed/33500419
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-020-20818-5
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