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Kv1.1 potassium channel subunit deficiency alters ventricular arrhythmia susceptibility, contractility, and repolarization
Epilepsy‐associated Kv1.1 voltage‐gated potassium channel subunits encoded by the Kcna1 gene have traditionally been considered absent in heart, but recent studies reveal they are expressed in cardiomyocytes where they could regulate intrinsic cardiac electrophysiology. Although Kv1.1 now has a demo...
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| Vydáno v: | Physiol Rep |
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| Hlavní autoři: | , , , , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
John Wiley and Sons Inc.
2021
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7798052/ https://ncbi.nlm.nih.gov/pubmed/33427415 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.14702 |
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