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ENaC expression correlates with the acute furosemide‐induced K(+) excretion
BACKGROUND: In the aldosterone‐sensitive distal nephron (ASDN), epithelial sodium channel (ENaC)‐mediated Na(+) absorption drives K(+) excretion. K(+) excretion depends on the delivery of Na(+) to the ASDN and molecularly activated ENaC. Furosemide is known as a K(+) wasting diuretic as it greatly e...
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| Publicado en: | Physiol Rep |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
John Wiley and Sons Inc.
2021
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7788322/ https://ncbi.nlm.nih.gov/pubmed/33410279 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.14668 |
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