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Redox and mTOR-dependent regulation of plasma lamellar calcium influx controls the senescence-associated secretory phenotype
Cellular senescence has evolved as a protective mechanism to arrest growth of cells with oncogenic potential but is accompanied by the often pathologically deleterious senescence-associated secretory phenotype (SASP). Here we demonstrate an H(2)O(2)-dependent functional disruption controlling senesc...
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| Publicat a: | Exp Biol Med (Maywood) |
|---|---|
| Autors principals: | , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
SAGE Publications
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7787549/ https://ncbi.nlm.nih.gov/pubmed/32686475 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/1535370220943122 |
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