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Development of experimental chronic kidney disease and vascular calcification alters diurnal variation of phosphate and its hormonal regulators
The mineral‐bone axis is tightly regulated and dependent on renal function. In chronic kidney disease (CKD) progressive loss of renal capacity disrupts this axis over‐time, with marked changes in circulating calcium, phosphate, PTH, and fibroblast growth factor‐23 (FGF‐23). These changes contribute...
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| Publicado no: | Physiol Rep |
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| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
John Wiley and Sons Inc.
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7666773/ https://ncbi.nlm.nih.gov/pubmed/33190417 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.14626 |
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