Development of experimental chronic kidney disease and vascular calcification alters diurnal variation of phosphate and its hormonal regulators

The mineral‐bone axis is tightly regulated and dependent on renal function. In chronic kidney disease (CKD) progressive loss of renal capacity disrupts this axis over‐time, with marked changes in circulating calcium, phosphate, PTH, and fibroblast growth factor‐23 (FGF‐23). These changes contribute...

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Bibliografiske detaljer
Udgivet i:Physiol Rep
Main Authors: Svajger, Bruno A., Riddoch, Justin L. H., Pruss, Cynthia M., Laverty, Kimberly J., Ward, Emilie, Holden, Rachel M., Adams, Michael A.
Format: Artigo
Sprog:Inglês
Udgivet: John Wiley and Sons Inc. 2020
Fag:
Original Research
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7666773/
https://ncbi.nlm.nih.gov/pubmed/33190417
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.14626
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