Development of experimental chronic kidney disease and vascular calcification alters diurnal variation of phosphate and its hormonal regulators

Abstract The mineral‐bone axis is tightly regulated and dependent on renal function. In chronic kidney disease (CKD) progressive loss of renal capacity disrupts this axis over‐time, with marked changes in circulating calcium, phosphate, PTH, and fibroblast growth factor‐23 (FGF‐23). These changes co...

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Main Authors: Bruno A. Svajger, Justin L. H. Riddoch, Cynthia M. Pruss, Kimberly J. Laverty, Emilie Ward, Rachel M. Holden, Michael A. Adams
Formato: Artigo
Idioma:Inglês
Publicado: Wiley 2020-11-01
Series:Physiological Reports
Assuntos:
CKD
diurnal
FGF‐23
phosphate
vascular calcification
Acceso en liña:https://doi.org/10.14814/phy2.14626
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