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Imaging Aβ(1–42) fibril elongation reveals strongly polarised growth and growth incompetent states†
The major hallmark of Alzheimer’s disease is the deposition of plaques of amyloid fibrils formed from amyloid-β (Aβ) peptides. Kinetic studies have contributed significantly towards a mechanistic understanding of amyloid fibril self-assembly, however dynamic features of the aggregation process canno...
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| Publicado no: | Phys Chem Chem Phys |
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| Main Authors: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7612976/ https://ncbi.nlm.nih.gov/pubmed/29026905 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1039/c7cp03412a |
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