The AKR1C3/AR‐V7 complex maintains CRPC tumour growth by repressing B4GALT1 expression

Multiple mechanisms contribute to the survival and growth of metastatic castration‐resistant prostate cancer (mCRPC) cells without androgen, including androgen receptor splice variants (AR‐V) and de novo intratumoral androgen synthesis. AKR1C3 is a critical androgenic enzyme that plays different rol...

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Detaylı Bibliyografya
Yayımlandı:J Cell Mol Med
Asıl Yazarlar: Wang, Bin, Wu, Shiqi, Fang, Yong, Sun, Guangxi, He, Dalin, Hsieh, Jer‐Tsong, Wang, Xinyang, Zeng, Hao, Wu, Kaijie
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: John Wiley and Sons Inc. 2020
Konular:
Original Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC7579719/
https://ncbi.nlm.nih.gov/pubmed/32902124
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.15831
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