RGFP966 inactivation of the YAP pathway attenuates cardiac dysfunction induced by prolonged hypothermic preservation

Oxidative stress and apoptosis are the key factors that limit the hypothermic preservation time of donor hearts to within 4–6 h. The aim of this study was to investigate whether the histone deacetylase 3 (HDAC3) inhibitor RGFP966 could protect against cardiac injury induced by prolonged hypothermic...

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Veröffentlicht in:J Zhejiang Univ Sci B
Hauptverfasser: Zheng, Xiao-he, Wang, Lin-lin, Zheng, Ming-zhi, Zhong, Jin-jie, Chen, Ying-ying, Shen, Yue-liang
Format: Artigo
Sprache:Inglês
Veröffentlicht: Zhejiang University Press 2020
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Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7519627/
https://ncbi.nlm.nih.gov/pubmed/32893527
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1631/jzus.B2000026
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