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S100B Impairs Oligodendrogenesis and Myelin Repair Following Demyelination Through RAGE Engagement

Increased expression of S100B and its specific receptor for advanced glycation end products (RAGE) has been described in patients with multiple sclerosis (MS), being associated with an active demyelinating process. We previously showed that a direct neutralization of S100B reduces lysophosphatidylch...

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Detalhes bibliográficos
Publicado no:Front Cell Neurosci
Main Authors: Santos, Gisela, Barateiro, Andreia, Brites, Dora, Fernandes, Adelaide
Formato: Artigo
Idioma:Inglês
Publicado em: Frontiers Media S.A. 2020
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7500156/
https://ncbi.nlm.nih.gov/pubmed/33100970
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2020.00279
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