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S100B Impairs Oligodendrogenesis and Myelin Repair Following Demyelination Through RAGE Engagement
Increased expression of S100B and its specific receptor for advanced glycation end products (RAGE) has been described in patients with multiple sclerosis (MS), being associated with an active demyelinating process. We previously showed that a direct neutralization of S100B reduces lysophosphatidylch...
שמור ב:
| הוצא לאור ב: | Front Cell Neurosci |
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| Main Authors: | , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Frontiers Media S.A.
2020
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7500156/ https://ncbi.nlm.nih.gov/pubmed/33100970 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2020.00279 |
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