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PRMT5 is upregulated by B cell receptor signaling and forms a positive feedback loop with PI3K/AKT in lymphoma cells

PRMT5, which regulates gene expression by symmetric dimethylation of histones and non-histone target proteins, is overexpressed and plays a pathogenic role in many cancers. In diffuse large B cell lymphoma (DLBCL), the mechanisms of PRMT5 dysregulation and its role in lymphomagenesis remain largely...

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מידע ביבליוגרפי
הוצא לאור ב:	Leukemia
Main Authors:	Zhu, Fen, Guo, Hui, Bates, Paul D., Zhang, Shanxiang, Zhang, Hui, Nomie, Krystle J., Li, Yangguang, Lu, Li, Seibold, Kaitlyn R., Wang, Fangyu, Rumball, Ian, Cameron, Hunter, Hoang, Nguyet M., Yang, David T., Xu, Wei, Zhang, Liang, Wang, Michael, Capitini, Christian M., Rui, Lixin
פורמט:	Artigo
שפה:	Inglês
יצא לאור:	2019
נושאים:	Article
גישה מקוונת:	https://ncbi.nlm.nih.gov/pmc/articles/PMC7494157/ https://ncbi.nlm.nih.gov/pubmed/31123343 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41375-019-0489-6
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PRMT5 is upregulated by B cell receptor signaling and forms a positive feedback loop with PI3K/AKT in lymphoma cells

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